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Al-Azhar University – Gaza
2025
Environmental Analysis Health and Toxicology
Astaxanthin ameliorates necroptosis through bisphenol-A exposure by regulating brain RIPK1/FADD/RIPK3/MLKL pathway in adult male rats
40
3
Korean Society of Environmental Health and Toxicology
e2025024
Department of Laboratory Medical Sciences, Al-Aqsa University, Mustafa Hafez St, Gaza, Palestine; Faculty of Medicine, Al Azhar University, Jamal Abdl Naser St, Gaza, Palestine; Mammalian Toxicology Department, Central Agricultural Pesticides Laboratory, Agricultural Research Center, Dokki, Giza, Egypt; Department of Chemistry, College of Science, King Khalid University, Abha, 61413, Saudi Arabia; Pesticide Formulation Department, Central Agricultural Pesticide Laboratory, Agricultural Research Center, Dokki, Giza, Egypt; Egyptian Center for Disease Control (CDC), National Hepatology and Tropical Medicine Research Institute (NHTMRI), Corniche El Nil-Imbaba, Giza, Egypt
1
10.5620/eaht.2025024
acetylcholinesterase; astaxanthin; bisphenol A; caspase 3; caspase 9; dopamine; Fas associated death domain protein; glutathione peroxidase; glutathione transferase; mixed lineage kinase; mixed lineage kinase domain like pseudokinase; neurotransmitter; protein bcl 2; protein kinase; protein p53; receptor interacting protein kinase 1; receptor interacting protein kinase 3; serotonin; superoxide dismutase; unclassified drug; adult; animal experiment; animal model; animal tissue; apoptosis; Article; controlled study; drug exposure; enzyme linked immunosorbent assay; gene expression; hippocampus; lipid peroxidation; male; necroptosis; nonhuman; oxidative stress; protein expression; rat; real time polymerase chain reaction; signal transduction